The kidneys play an important role in the composition and volume of the blood, the excretion of metabolic wastes in the urine, the control of acid/base balance in the body, and the production of hormones to maintain hemostasis. GBM damage in the glomeruli impairs the filtration process that allows proteins and red blood cells to leak into the urine. The loss of proteins such as albumin in the urine results in a decrease in their level in the bloodstream. Accordingly, this patient's blood reveals a reduced albumin (Alb) value of 2.9 g/dL, a reduced total serum protein value of 5.0 g/dL, and urine contains protein and red blood cells. Reduced filtering capacity results in the inability of the kidneys to excrete excretory products such as electrolytes and metabolic waste products which then accumulate in the blood. Furthermore, the inability of the distal convoluted tubules to excrete sufficient quantities of potassium, sodium, magnesium (Mg), chloride (Cl), urea, creatinine (Cr), alkaline phosphatase (Alk Phos) and phosphate (PO4) leads to increased distal convoluted tubules. the blood. His laboratory values ​​reveal an increase in sodium value of 149 meq/L, an increase in potassium value of 5.4 meq/L, an increase in chloride value of 116 meq/L, an increase in blood urea nitrogen in blood (BUN) in serum of 143 mg/dL and increase in serum creatinine of 7.14 mg/dl. The other abnormal blood tests associated with the loss of filtration properties of the kidneys found in this patient are related to an increase in the alkaline phosphatase value of 178 IU/L, an increase in the magnesium value of 3.8 mgdL and an increase in phosphate (PO4) value of 5.9 mg / dL. An increase in blood glucose of 152 mg/dL could be associated with an increase in uremia-induced insulin resistance. An increase in blood sugar could be explained by the presence of urea nitrogen in the blood equal to 143 mg/dl, higher than the normal limit of 20